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The Litlle Old Lady From Passadena jan & dean
Jan and Dean were a rock and roll duo, popular from the late 1950s through the mid 1960s, consisting (More) Jan and Dean were a rock and roll duo, popular from the late 1950s through the mid 1960s, consisting of William Jan Berry (April 3, 1941 March 26, 2004) and Dean Ormsby Torrence (born March 10, 1940). Although Jan & Dean pre-dated The Beach Boys, they became most famously associated with the vocal "surf music" craze inspired by The Beach Boys Jan Berry and Dean Torrence, both born in Los Angeles, California, began singing together as a duo after football practice at University High School. Primitive recording sessions followed soon after, in a makeshift studio in Berry's garage. They first performed onstage as "The Barons" at a high school dance. With the Barons, Jan Berry was experimenting with multi-part vocal arrangements five years before he started working professionally with Brian Wilson.[1] Their first commercial success was "Jennie Lee" (1958), which reached #8, and was an ode to a local, Hollywood burlesque performer that Jan Berry recorded with fellow Baron Arnie Ginsburg. "Jan & Arnie" released three singles in all. After Torrence returned from a stint in the army reserves, Jan Berry and Dean Torrence began to make music as "Jan and Dean". With the help of record producers Herb Alpert and Lou Adler, Jan and Dean scored a #10 hit with "Baby Talk" (1959), and then scored a series of hits over the next couple of years. Playing local venues, they met and performed with the Beach Boys, and discovered the appeal of the latter's "surf sound". By this time, Berry was co-writing, arranging, and producing all of Jan and Dean's original material. Berry signed a series of contracts with Screen Gems to write and produce music for Jan and Dean, as well as other artists such as Judy & Jill (which included Berry's girlfriend Jill Gibson and Dean Torrence's girlfriend Judy Lovejoy), The Matadors, and Pixie (a young female solo singer).[2] During this time, Berry co-wrote and/or arranged and produced songs for artists outside of Jan and Dean, including The Angels ("I Adore Him", Top 30), the Gents, the Matadors (Sinners), Judy & Jill, Pixie (unreleased), Jill Gibson, Shelley Fabares, Deane Hawley, the Rip Chords ("Three Window Coupe", Top 30), and Johnny Crawford, among others. [edit] Part-time musicians Unlike most other rock 'n roll acts of the period, Jan and Dean did not give music their full-time attention. Jan & Dean were college students, maintaining their studies while writing and recording music and making public appearances on the side. Torrence majored in advertising design in the school of architecture at USC. Berry took science and music classes at UCLA, and entered the California College of Medicine (now the UC Irvine School of Medicine) in 1963. By the time of his 1966 auto accident, Berry had completed two years of medical school.[3] [edit] Surf's golden boys: 1963-1964 Jan and Dean reached their commercial peak in 1963 and 1964. The duo scored an impressive sixteen Top 40 hits on the Billboard and Cash Box magazine charts, with a total of twenty-six chart hits over an eight-year period (1958-1966). Jan and Brian Wilson collaborated on roughly a dozen hits and album cuts for Jan and Dean, including the number one national hit "Surf City" in 1963. Subsequent top 10 hits included "Drag City" (#10) (1963), "The Little Old Lady from Pasadena" (#3) (1964), and the eerily portentous "Dead Man's Curve" (#8) (1964). In 1964, at the height of their fame, Jan and Dean hosted and performed at The T.A.M.I. Show, a historic concert film directed by Steve Binder. The film also featured such acts as The Rolling Stones, Chuck Berry, Gerry & The Pacemakers, James Brown, Billy J. Kramer & The Dakotas, Marvin Gaye, The Supremes, Lesley Gore, Smokey Robinson & The Miracles, and the Beach Boys (whose sequence was later cut from the film, due to contract violation issues). Also in 1964, the duo performed the title track for the Columbia Pictures film Ride the Wild Surf, starring Fabian, Tab Hunter, Peter Brown, Shelley Fabares, and Barbara Eden. The song, penned by Jan Berry, Brian Wilson, and Roger Christian, was a Top 20 national hit. Jan and Dean also filmed two unreleased television pilots: Surf Scene in 1963 and On the Run in 1966. Their feature film Easy Come, Easy Go was canceled when Berry, as well as the film's director and other crew members, were seriously injured in a railroad accident while shooting the movie in August 1965. [edit] Changing times: 1965-1966 After the surf craze, Jan and Dean scored two Top-30 hits in 1965: "You Really Know How to Hurt a Guy" and "I Found a Girl" the latter from the album Folk 'n Roll. During this period, they also began to experiment with cutting-edge comedy concepts such as the original (unreleased) Filet of Soul and Jan & Dean Meet Batman. The former's album cover shows Berry with his leg in a cast as a result of the accident while filming "Easy Come, Easy Go". [edit] Berry's car wreck and its aftermath: 1966-1968 On April 12, 1966, Berry received severe head injuries in a motor vehicle accident just a short distance from Dead Man's Curve in Los Angeles, California, two years after the song had become a hit. Berry was on his way to a business meeting when he crashed his Corvette into a parked truck on Whittier Drive in Beverly Hills. Berry had also separated from his girlfriend of seven years, singer-artist Jill Gibson, later a member for a short time of The Mamas and the Papas, who had also co-written several songs with Berry. Berry traveled a long and difficult road toward recovery from brain damage and partial paralysis. He had minimal use of his right arm, and had to learn to write with his left hand. Doctors said he would never walk again, but he refused to give up, and ultimately succeeded. Torrence stood by his partner, maintaining their presence in the music industry, and keeping open the possibility that they would perform together again.[4] In Berry's absence, Torrence released several singles on the J&D Record Co. label and recorded Save for a Rainy Day in 1966, a concept album featuring all rain-themed songs. Torrence posed with Berry's brother Ken for the album cover photos. Columbia Records released one single from the project ("Yellow Balloon") as did the song's writer, Gary Zekley, with The Yellow Balloon, but with legal wrangles scuttling Torrence's Columbia deal and Berry's disapproval of the project, Save for a Rainy Day remained a self-released album on the J&D Record Co. label.[5] Besides his studio work, Torrence became a graphic artist while Berry recovered, starting his own company, Kittyhawk Graphics, and designing and creating album covers and logos for other musicians and recording artists, including Harry Nilsson, Steve Martin, the Nitty Gritty Dirt Band, Dennis Wilson, Bruce Johnston, The Beach Boys, Diana Ross and The Supremes, Linda Ronstadt, Papa Doo Run Run, Canned Heat, The Ventures and many others. Torrence (with Gene Brownell) won a Grammy Award for Album Cover of the Year, for the group Pollution in 1973. Berry returned to the studio in April 1967, one year to the month after his accident. Working with collaborators, he began writing and producing music again. In December 1967, Jan and Dean signed an agreement with Warner Bros. Records. Warner issued two singles under the name Jan and Dean, but a 1968 Berry-produced album for Warner Bros., the psychedelic Carnival of Sound, remains unreleased.[6] [edit] Further progress: 1969-1978 Berry began to sing again in the early 1970s, and he arranged and produced a number of singles (both solo and as Jan & Dean) between 1972 and 1978 on the Ode and A&M labels, facilitated by friend and former manager Lou Adler.[7]Berry also toured with his Aloha band, while Dean began performing with a band called Papa Doo Run Run. In 1973, Jan & Dean made an appearance at the Hollywood Palladium, as part of Jim Pewter's "Surfer's Stomp" reunion. But the duo's first performance after Berry's accident backed with live musicians occurred at the Palomino Nightclub in North Hollywood, June 5, 1976 (ten years after the accident). Backing the duo was Dean's band, Papa Doo Run Run. The day after that performance there was a very positive review in "Variety" and the phones started ringing. Promoters everywhere wanted to hire Jan & Dean. So in the fall of 1976 a mini-tour of the Pacific Northwest was planned with Jan & Dean backed by Papa Doo Run Run. The tour was a rousing success. J & D then hired a manager and agency representation, and between 1977 and 1980, there were 4 nationwide J & D tours with Papa Doo as their opening act and backup band - sell out crowds from coast to coast. Even though a dozen years had passed, Jan was still suffering the effects of his horrific accident, with partial paralysis and aphasia. But Jan's limp and useless right arm, plus his difficulty in speaking were totally overlooked by his adoring fans. Jan Berry was back! [8] [9] In 1974, attorney Paul Morantz published a landmark article about Jan Berry's recovery in Rolling Stone magazine.[10] [edit] Back on the road: 1978-2004 On February 3, 1978, CBS aired a made-for-TV movie about the duo titled Deadman's Curve. The biopic starred Richard Hatch as Jan Berry and Bruce Davison as Dean Torrence, with cameo appearances by Dick Clark, Wolfman Jack, Mike Love of the Beach Boys, and Bruce Johnston (who at that time was temporarily out of the Beach Boys), as well as Berry himself (near the end of the movie, he can be seen sitting in the audience, watching "himself" (Richard Hatch) perform onstage). The part of Jan & Dean's band, Papa Doo Run Run, was played by themselves. Johnston and Berry had known each other since high school, and had played music together in Berry's garage in Bel Air long before Jan & Dean or the Beach Boys were formed. Following the release of the film, the duo made steps toward an official comeback that year, including touring with the Beach Boys. In the early 1980s, Papa Doo Run Run left the duo to explore other performance and recording ventures. Berry struggled to overcome drug addiction, so Torrence toured briefly as "Mike & Dean", with Mike Love of the Beach Boys. Once Berry got sober, beating the odds once again, the duo reunited for good. In "Phase II" of their career, Dean Torrence led the touring operation. In 1986, Berry helped establish the Jan Berry Center for the Brain Injured in Downey, California. Dean Torrence participated in the promotional campaign for this endeavor. Though Berry only made a partial recovery, he persevered and remained a high-profile example for patients with traumatic brain injury.[11] Jan and Dean continued to tour on their own throughout the 1980s, 1990s, and into the new millennium with 1960s nostalgia providing them with a ready audience. Sundazed Records reissued Save for a Rainy Day in 1996, and the album drew critical praise.[citation needed] Between the 1970s and 1990s, Torrence issued a number of re-recordings of classic Jan and Dean hits. An album titled One Summer Night / Live was issued by Rhino Records in 1982, and Dean collaborated with Berry on Port to Paradise, released on J&D Records in 1986. In 1997, after many years of hard work, Berry released a solo album called Second Wave on One Way Records. On August 31, 1991, Berry married Gertie Filip at The Stardust Convention Centre in Las Vegas, Nevada. Torrence was Berry's best man at the wedding. Jan and Dean ended with Jan Berry's death on March 26, 2004, at the age of 62. Berry was an organ donor, and his body was cremated. On April 18, 2004, a "Celebration of Life" was held in Berry's memory at The Roxy Theatre on the Sunset Strip in West Hollywood, California. Celebrities attending the event included Dean Torrence, Lou Adler, Jill Gibson, and Nancy Sinatra. Also present were many family members, friends, and musicians associated with Jan and Dean and the Beach Boys including the original 1970's version of Papa Doo Run Run. Torrence now tours occasionally with The Surf City Allstars. He serves as a spokesman for the City of Huntington Beach California, which, thanks in part to his efforts, is nationally recognized as "Surf City USA". He officially endorses the Official Jan & Dean Fan Site. Dean Torrence lives in Huntington Beach, California with his wife and two daughters. Katie Torrence, his oldest daughter, is reportedly recording an album.[citation needed] [edit] Jan and Dean's place in rock history According to rock critic Dave Marsh, the attitude and public persona of punk rock can be traced to Jan and Dean.[12] Moreover, both Jan Berry and Dean Torrence's anti-establishment attitude toward the music industry is well documented.[citation needed] Their music has been covered by numerous Punk and alternative bands since the 1970s. Along with Phil Spector, Brian Wilson, and Lee Hazlewood, Berry enjoyed a reputation as one of the best record producers on the West Coast.[13] Brian Wilson has cited Berry as having a direct impact on his own growth as a record producer.[14] Dean Torrence believes the group should be in the Rock & Roll Hall of Fame: "We have the scoreboard if you just want to compare number of hits and musical projects done. We beat 75 percent of the people in there. So what else is it? I've got to think that we were pretty irreverent when it came to the music industry. They kind of always held that against us. That's OK with me."[ (Less)
3-6 Microvascular Decompression MVD Dr. Parrish Neurosurgeon
Click More http://www.MyTrigeminalNeuralgiaStory.com AWC 4398 3-6 Microvascular Decompression MVD (More) Click More http://www.MyTrigeminalNeuralgiaStory.com AWC 4398 3-6 Microvascular Decompression MVD Click Dr.Parrish Neurosurgeon TN Tic douloureux Facial Pain Electric Shocks. TNA BrianNelson123 Suicide Painful Jannetta Association Teflon Nerve THIS WEBSITE IS DESIGNED TO HAVE EACH TRIGEMINAL NEURALGIA patient tell there story from the beginning of the problem to the current status which is understandably changing daily as the body processes more of the pain. My personal story is very long and and be seen at w htttp[://www.IamFightingCancer.com Important words found on this site. Trigeminal Neuralgia Minneapolis TN Pain Personal Story, Balloon Compression Mentor, dysesthesia, bad feeling constant spasm. excruciating pains, Henry, Pneumonia Electrical Shocks, Shirley, Shelly Wilson, Support Group, Education, Association, Stabbing, Jolts, Suicide Disease, Neuropathic, rare Disorder, Treatment, destructive surgery, Procedure, Microvascular Decompression, tic douloureux Marge Prietz Trigeminal Neuralgia Extreme Facial Pain TN Websites insert. YouTube. From NelsonIdeas.com Trigeminal Neuralgia Extreme Facial Pain TN Websites insert. Websites insert. My Trigeminal Neuralgia Extreme Facial Pain TN Websites http:/./www.NelsonIdeas.com Click Dental Education Trigeminal Neuralgia Extreme Facial Pain http://www.NewMedicalDirectories.com/Trigeminal-Neuralgia-Dental/Dentist-Dentists.html Click Trigeminal Neuralgia Patient Painful-Stories http://www.BrianNelsonConsulting.com/trigeminal-neuralgia-tn/patient-painful-stories.html Click My Trigeminal Neuralgia (TN) Story only http://www.PartyTentCity.com/mytnstory.html Click My Story on TN Brian N http://www.PartyTentCity.com/trigeminal-neuralgia-tn-tmj-my-story/directory.html Click Trigeminal Neuralgia Slide Show Story of Pain http://www.NewMedicalDirectories.com/Trigeminal-Neuralgia-Slide-Show/Draft.html Click Medical Data Base Medical Costs More Expensive Due to Non Use of Technology http://www.briannelsonconsulting.com/medical-data-base/faq-info.html Click MyTrigeminal Neuralgia Story Directory http://www.MyTrigeminalNeuralgiaStory.com Click Slide Show Draft for New TN Patients. http://www.newmedicaldirectories.com/Trigeminal-Neuralgia-Slide-Show/Draft.html Click-Trigeminal Neuralgia Assn Page 1 http://newmedicaldirectories.com/Trigeminal-Neuralgia-Association/TN-Facial-Pain.html Click-Trigeminal Neuralgia Assn Page 2 http://newmedicaldirectories.com/Trigeminal-Neuralgia-Association/TN-Facial-Pain-2.html Click What is Trigeminal Neuragia? Portland,OR Slide Show http://www.NewMedicalDirectories.com/Trigeminal-Neuralgia-Slide-Show/Draft.html Click Trigeminal Neuralgia National Conference http://www.NewMedicalDirectories.com/Trigeminal-Neuralgia-Slide-Show/Draft.html Click Trigeminal Neuralgia Brian's Journal Tic Douloureux (TN) FacialPain-Cancer http://www.BrianNelsonConsulting.com/trigeminal-neuralgia-tn/faq-info.html http://www.BrianNelsonConsulting.com/trigeminal-neuralgia-tn/faq-info.html Click Page 1. Trigeminal Neuralgia http://www.BrianNelsonConsulting.com/trigeminal-neuralgia-tn/faq-info.html Click Page 2 Trigeminal Neuralgia http://www.briannelsonconsulting.com/trigeminal-neuralgia-tn/faq-info2.html Click Page 3 Trigeminal Neuralgia http://www.briannelsonconsulting.com/trigeminal-neuralgia-tn/faq-info3.htm Click Page 4 Trigeminal Neuralgia http://www.BrianNelsonConsulting.com/trigeminal-neuralgia-tn/faq-info4.html Click MyTrigeminal Neuralgia Stories Directory http://www.MyTrigeminalNeuralgiaStory.com/Index.html Click Brian's TN Story Quck Version http://www.MyTrigeminalNeuralgiaStory.com/BrianNelson/TN1.html Click Shirley's Story Trigeminal Neuralgia http://www.MyTrigeminalNeuralgiaStory.com/ShirleyH/TN3.html Click Sand's Story TN WHAT IS TRIGEMINAL NEURALGIA? TN (Trigeminal Neuralgia) is a pain that is described as among the most acute known to mankind. TN produces excruciating, lightning strikes of facial pain, typically near the nose, lips, eyes or ears. It is a disorder of the trigeminal nerve, which is the fifth and largest cranial nerve. TN (Trigeminal Neuralgia / tic douloureux) is a disorder of the fifth cranial (trigeminal) nerve that causes episodes of intense, stabbing, electric shock-like pain in the areas of the face where the branches of the nerve are distributed - lips, eyes, nose, scalp, forehead, upper jaw, and lower jaw. By many, it's called the "suicide disease". A less common form of the disorder called "Atypical Trigeminal Neuralgia" may cause less intense, constant, dull burning or aching pain, sometimes with occasional electric shock-like stabs. Both forms of the disorder most often affect one side of the face, but some patients experience pain at different times on both sides. Onset of symptoms occurs most often after age 50, but cases are known in children and even infants. Something as simple and routine as brushing the teeth, putting on makeup or even a slight breeze can trigger an attack, resulting in sheer agony for the individual. Trigeminal neuralgia (TN) is not fatal, but it is universally considered to be the most painful affliction known to medical practice. Initial treatment of TN is usually by means of anti-convulsant drugs, such as Tegretol or Neurontin. Some anti-depressant drugs also have significant pain relieving effects. Should medication be ineffective or if it produces undesirable side effects, neurosurgical procedures are available to relieve pressure on the nerve or to reduce nerve sensitivity. Some patients report having reduced or relieved pain by means of alternative medical therapies such as acupuncture, chiropractic adjustment, self-hypnosis or meditation. http://www.MyTrigeminalNeuralgiaStory.com/SandiW/TN4.html What is Trigeminal Neuralgia? Trigeminal neuralgia (TN), also called tic douloureux, is a chronic pain condition that causes extreme, sporadic, sudden burning or shock-like face pain that lasts anywhere from a few seconds to as long as 2 minutes per episode. The intensity of pain can be physically and mentally incapacitating. TN pain is typically felt on one side of the jaw or cheek. Episodes can last for days, weeks, or months at a time and then disappear for months or years. In the days before an episode begins, some patients may experience a tingling or numbing sensation or a somewhat constant and aching pain. The attacks often worsen over time, with fewer and shorter pain-free periods before they recur. The intense flashes of pain can be triggered by vibration or contact with the cheek (such as when shaving, washing the face, or applying makeup), brushing teeth, eating, drinking, talking, or being exposed to the wind. TN occurs most often in people over age 50, but it can occur at any age, and is more common in women than in men. There is some evidence that the disorder runs in families, perhaps because of an inherited pattern of blood vessel formation. Although sometimes debilitating, the disorder is not life-threatening. The presumed cause of TN is a blood vessel pressing on the trigeminal nerve in the head as it exits the brainstem. TN may be part of the normal aging process but in some cases it is the associated with another disorder, such as multiple sclerosis or other disorders characterized by damage to the myelin sheath that covers certain nerves. Is there any treatment? Because there are a large number of conditions that can cause facial pain, TN can be difficult to diagnose. But finding the cause of the pain is important as the treatments for different types of pain may differ. Treatment options include medicines such as anticonvulsants and tricyclic antidepressants, surgery, and complementary approaches. Typical analgesics and opioids are not usually helpful in treating the sharp, recurring pain caused by TN. If medication fails to relieve pain or produces intolerable side effects such as excess fatigue, surgical treatment may be recommended. Several neurosurgical procedures are available. Some are done on an outpatient basis, while others are more complex and require hospitalization. Some patients choose to manage TN using complementary techniques, usually in combination with drug treatment. These techniques include acupuncture, biofeedback, vitamin therapy, nutritional therapy, and electrical stimulation of the nerves. What is the prognosis? The disorder is characterized by recurrences and remissions, and successive recurrences may incapacitate the patient. Due to the intensity of the pain, even the fear of an impending attack may prevent activity. Trigeminal neuralgia is not fatal. What research is being done? Within the NINDS research programs, trigeminal neuralgia is addressed primarily through studies associated with pain research. NINDS vigorously pursues a research program seeking new treatments for pain and nerve damage with the ultimate goal of reversing debilitating conditions such as trigeminal neuralgia. NINDS has notified research investigators that it is seeking grant applications both in basic and clinical pain research. An Alternate Strategy Instead of waiting for the pain to become intractable or the medications toxic, an individual with trigeminal neuralgia has the option to request early surgery. This has a number of potential advantages: • Avoid years of medication and intermittent pain • Avoid facing surgery when old or infirm • If the person has a vascular loop, early microvascular decompression will increase the possibility of a successful operation with decreased risk of recurrence (evidence suggests better outcomes and lower recurrence rate the shorter the interval between onset of symptoms and nerve decompression) How To Find Out If You Have a Vascular Loop The conventional MRI scans used to rule out the presence of a brain tumor or multiple sclerosis as a cause of a patients face pain are not adequate to visualize the trigeminal nerve or an associated blood vessel. Fortunately, the continued improvement in MRI neuro-imaging now makes it possible to visualize both. The technique, which is called 3-D volume acquisition, is performed with contrast injection and utilizes thin cuts (0.8mm), without gaps similar to what was developed for MRI angiography and venography. The trigeminal nerve is easily visualized in the axial plane when the MRI series is centered at the midpoint of the fourth ventricle. To ensure an adequate evaluation, the nerve should be seen on three adjacent cuts. Early studies indicate that when an offending vessel is present it will be detected 80% of the of the time. With continued imaging improvements this percentage will definitely increase. Click here for UCSD Trigeminal Neuralgia Sequence Parameters for Seimens and GE MR Scanners. Surgical Options: Non-Destructive Procedures The only non-destructive procedure which reliably relieves the symptoms of Trigeminal Neuralgia is Microvascular Decompression (MVD). This involves surgical exploration with the operating microscope and visualization of the junction where the Trigeminal nerve enters the base of the brain, followed by coagulation or moving and padding away any compressing blood vessels. The advantage is pain relief without numbness in the majority of patients, which usually lasts indefinitely. If the pain recurs after a MVD, which it does in 10-15% of patients, it can usually be controlled with low dose Tegretol® or Neurontin®. If the pain continues, it will require a repeat MVD or one of the destructive procedures. Surgical Options: Destructive Procedures There are multiple destructive procedures which are beneficial in the treatment of Trigeminal Neuralgia. The most common of which are glycerol injections, gamma knife radiation, electrocoagulation, and balloon compression. These procedures are all based on interrupting the pain by partial damage to Trigeminal nerve fibers. Generally the more numbness they produce, the longer they last. The specific advantages and disadvantages need to be discussed with the surgeon performing the procedure. These procedures are recommended for patients who have failed MVD or are not candidates for major surgery. Comments Treatment is always individualized. All of the options above should be considered in consultation with a neurosurgeon familiar in their use. Recommendations Based on the data currently available, and in an effort to maximize quality of life, we recommend the following: Patients with less than 10 year life expectancy Refer for destructive procedure if pain not controlled medically without significant side effects Patients with more than 10 but less than 20 year life expectancy Consider destructive procedure May abolish need for continued increasing medications Will make medical therapy easier even if fails Patients with more than 20 year life expectancy Perform thin cut MRI with 3-D Volume Acquisition If vessel present recommend MVD 25 ARTICLE SECTIONS From the Mayo Clinic. Trigeminal neuralgia http://www.mayoclinic.com/health/trigeminal-neuralgia/DS00446 Introduction Signs and symptoms Causes When to seek medical advice Screening and diagnosis Treatment Coping skills Introduction Imagine having a jab of lightning-like pain shoot through your face when you brush your teeth or put on makeup. Sound excruciating? If you have trigeminal neuralgia, attacks of such pain are frequent and can often seem unbearable. You may initially experience short, mild attacks, but trigeminal neuralgia can progress, causing longer, more frequent bouts of searing pain. These painful attacks can be spontaneous, but they may also be provoked by even mild stimulation of your face, including brushing your teeth, shaving or putting on makeup. The pain of trigeminal neuralgia may occur in a fairly small area of your face, or it may spread rapidly over a wider area. Because of the variety of treatment options available, having trigeminal neuralgia doesn't necessarily mean you're doomed to a life of pain. Doctors usually can effectively manage trigeminal neuralgia, either with medications or surgery. Signs and symptoms An attack of trigeminal neuralgia can last from a few seconds to about a minute. Some people have mild, occasional twinges of pain, while other people have frequent, severe, electric-shock-like pain. The condition tends to come and go. You may experience attacks of pain off and on all day, or even for days or weeks at a time. Then, you may experience no pain for a prolonged period of time. Remission is less common the longer you have trigeminal neuralgia. People who have experienced severe trigeminal neuralgia have described the pain as: Lightning-like or electric-shock-like Shooting Jabbing Like having live wires in your face Trigeminal neuralgia usually affects just one side of your face. The pain may affect just a portion of one side of your face or spread in a wider pattern. Rarely, trigeminal neuralgia can affect both sides of your face, but not at the same time. Causes Branches of the trigeminal nerve CLICK TO ENLARGE The condition is called trigeminal neuralgia because the painful facial areas are those served by one or more of the three branches of your trigeminal nerve. This large nerve originates deep inside your brain and carries sensation from your face to your brain. The pain of trigeminal neuralgia is due to a disturbance in the function of the trigeminal nerve. Trigeminal neuralgia is also known as tic douloureux. The cause of the pain usually is due to contact between a normal artery or vein and the trigeminal nerve at the base of your brain. This places pressure on the nerve as it enters your brain and causes the nerve to misfire. Physical nerve damage or stress may be the initial trigger for trigeminal neuralgia. After the trigeminal nerve leaves your brain and travels through your skull, it divides into three smaller branches, controlling sensation throughout your face: The first branch controls sensation in your eye, upper eyelid and forehead. The second branch controls sensation in your lower eyelid, cheek, nostril, upper lip and upper gum. The third branch controls sensations in your jaw, lower lip, lower gum and some of the muscles you use for chewing. You may feel pain in the area served by just one branch of the trigeminal nerve, or the pain may affect all branches on one side of your face. Besides compression from blood vessel contact, other less frequent sources of pain to the trigeminal nerve may include: Compression by a tumor Multiple sclerosis A stroke affecting the lower part of your brain, where the trigeminal nerve enters your central nervous system A variety of triggers, many subtle, may set off the pain. These triggers may include: Shaving Stroking your face Eating Drinking Brushing your teeth Talking Putting on makeup Encountering a breeze Smiling Trigeminal neuralgia affects women more often than men. The disorder is more likely to occur in people who are older than 50. About 5 percent of people with trigeminal neuralgia have other family members with the disorder, which suggests a possible genetic cause in some cases. When to seek medical advice Some people mistake the pain of trigeminal neuralgia for a toothache or a headache. It's not uncommon for people to believe that their facial pain is dental-related, particularly when the pain seems to stem from the gumline or is located near a tooth. If you experience facial pain, particularly prolonged pain or pain that hasn't gone away with use of over-the-counter pain relievers, see your dentist or doctor. Screening and diagnosis If you go to your dentist, an examination of your mouth can reveal whether a problem with your teeth or gums is causing your pain. If you go to your doctor, he or she will want to ask about your medical history and have you describe your pain — how severe it is, what part of your face it affects, how long pain lasts and what seems to trigger episodes of pain. You'll also undergo a neurologic examination. During this examination, your doctor examines and touches parts of your face to try to determine exactly where the pain is occurring and — if it appears that you have trigeminal neuralgia — which branches of the trigeminal nerve may be affected. Your doctor may exclude other possible conditions based on your medical history, the examination, and a magnetic resonance imaging (MRI) scan of your head. Treatment Medications are the usual initial treatment for trigeminal neuralgia. Medications are often effective in lessening or blocking the pain signals sent to your brain. A number of drugs are available. If you stop responding to a particular medication or experience too many side effects, switching to another medication may work for you. Medications Carbamazepine (Tegretol, Carbatrol). Carbamazepine, an anticonvulsant drug, is the most common medication that doctors use to treat trigeminal neuralgia. In the early stages of the disease, carbamazepine controls pain for most people. However, the effectiveness of carbamazepine decreases over time. Side effects include dizziness, confusion, sleepiness and nausea. Baclofen. Baclofen is a muscle relaxant. Its effectiveness may increase when it's used in combination with carbamazepine or phenytoin. Side effects include confusion, nausea and drowsiness. Phenytoin (Dilantin, Phenytek). Phenytoin, another anticonvulsant medication, was the first medication used to treat trigeminal neuralgia. Side effects include gum enlargement, dizziness and drowsiness. Oxcarbazepine (Trileptal). Oxcarbazepine is another anticonvulsant medication and is similar to carbamazepine. Side effects include dizziness and double vision. Doctors may sometimes prescribe other medications, such as lamotrignine (Lamictal) or gabapentin (Neurontin). Some people with trigeminal neuralgia eventually stop responding to medications, or they experience unpleasant side effects. For those people, surgery, or a combination of surgery and medications, may be an option. Surgery The goal of a number of surgical procedures is to either damage or destroy the part of the trigeminal nerve that's the source of your pain. Because the success of these procedures depends on damaging the nerve, facial numbness of varying degree is a common side effect. These procedures involve: Alcohol injection. Alcohol injections under the skin of your face, where the branches of the trigeminal nerve leave the bones of your face, may offer temporary pain relief by numbing the areas for weeks or months. Because the pain relief isn't permanent, you may need repeated injections or a different procedure. Glycerol injection. This procedure is called percutaneous glycerol rhizotomy (PGR). "Percutaneous" means through the skin. Your doctor inserts a needle through your face and into an opening in the base of your skull. The needle is guided into the trigeminal cistern, a small sac of spinal fluid that surrounds the trigeminal nerve ganglion (the area where the trigeminal nerve divides into three branches) and part of its root. Images are made to confirm that the needle is in the proper location. After confirming the location, your doctor injects a small amount of sterile glycerol. After three or four hours, the glycerol damages the trigeminal nerve and blocks pain signals. Initially, PGR relieves pain in most people. However, some people have a recurrence of pain, and many experience facial numbness or tingling. http://www.MyTrigeminalNeuralgiaStory.com Balloon compression. In a procedure called percutaneous balloon compression of the trigeminal nerve (PBCTN), your doctor inserts a hollow needle through your face and into an opening in the base of your skull. Then, a thin, flexible tube (catheter) with a balloon on the end is threaded through the needle. The balloon is inflated with enough pressure to damage the nerve and block pain signals. PBCTN successfully controls pain in most people, at least for a while. Most people undergoing PBCTN experience facial numbness of varying degrees, and more than half experience nerve damage resulting in a temporary or permanent weakness of the muscles used to chew. http://www.MyTrigeminalNeuralgiaStory.com Electric current. A procedure called percutaneous stereotactic radiofrequency thermal rhizotomy (PSRTR) selectively destroys nerve fibers associated with pain. Your doctor threads a needle through your face and into an opening in your skull. Once in place, an electrode is threaded through the needle until it rests against the nerve root. An electric current is passed through the tip of the electrode until it's heated to the desired temperature. The heated tip damages the nerve fibers and creates an area of injury (lesion). If your pain isn't eliminated, your doctor may create additional lesions. PSRTR successfully controls pain in most people. Facial numbness is a common side effect of this type of treatment. The pain may return after a few years. Microvascular decompression (MVD). A procedure called microvascular decompression (MVD) doesn't damage or destroy part of the trigeminal nerve. Instead, MVD involves relocating or removing blood vessels that are in contact with the trigeminal root and separating the nerve root and blood vessels with a small pad. During MVD, your doctor makes an incision behind one ear. Then, through a small hole in your skull, part of your brain is lifted to expose the trigeminal nerve. If your doctor finds an artery in contact with the nerve root, he or she directs it away from the nerve and places a pad between the nerve and the artery. Doctors usually remove a vein that is found to be compressing the trigeminal nerve. MVD can successfully eliminate or reduce pain most of the time, but as with all other surgical procedures for trigeminal neuralgia, pain can recur in some people. http://www.MyTrigeminalNeuralgiaStory.com While MVD has a high success rate, it also carries risks. There are small chances of decreased hearing, facial weakness, facial numbness, double vision, and even a stroke or death. The risk of facial numbness is less with MVD than with procedures that involve damaging the trigeminal nerve. Severing the nerve. A procedure called partial sensory rhizotomy (PSR) involves cutting part of the trigeminal nerve at the base of your brain. Through an incision behind your ear, your doctor makes a quarter-sized hole in your skull to access the nerve. This procedure usually is helpful, but almost always causes facial numbness. And it's possible for pain to recur. If your doctor doesn't find an artery or vein in contact with the trigeminal nerve, he or she won't be able to perform an MVD, and a PSR may be done instead. Radiation. Gamma-knife radiosurgery (GKR) involves delivering a focused, high dose of radiation to the root of the trigeminal nerve. The radiation damages the trigeminal nerve and reduces or eliminates the pain. Relief isn't immediate and can take several weeks to begin. GKR is successful in eliminating pain more than half of the time. Sometimes the pain may recur. The procedure is painless and typically is done without anesthesia. Because this procedure is relatively new, the long-term risks of this type of radiation are not yet known. • Coping skills Living with trigeminal neuralgia can be difficult. The disorder may affect your interaction with friends and family, your productivity at work, and the overall quality of your life. You may find that talking to a counselor or therapist can help you cope with the effects of trigeminal neuralgia, or you may find encouragement and understanding in a support group. Although support groups aren't for everyone, they can be good sources of information. Group members often know about the latest treatments and tend to share their own experiences. If you're interested, your doctor may be able to recommend a group in your area. 27 Background: Trigeminal neuralgia (TN), also known as tic douloureux, is a pain syndrome recognizable by patient history alone. The condition is characterized by pain often accompanied by a brief facial spasm or tic. Pain distribution is unilateral and follows the sensory distribution of cranial nerve V, typically radiating to the maxillary (V2) or mandibular (V3) area. At times, both distributions are affected. Physical examination eliminates alternative diagnoses. Signs of cranial nerve dysfunction or other neurologic abnormality exclude the diagnosis of idiopathic TN and suggest that pain may be secondary to a structural lesion. Pathophysiology: The mechanism of pain production remains controversial. One theory suggests that peripheral injury or disease of the trigeminal nerve increases afferent firing in the nerve; failure of central inhibitory mechanisms may be involved as well. Pain is perceived when nociceptive neurons in a trigeminal nucleus involve thalamic relay neurons. Aneurysms, tumors, chronic meningeal inflammation, or other lesions may irritate trigeminal nerve roots along the pons. An abnormal vascular course of the superior cerebellar artery is often cited as the cause. In most cases, no lesion is identified, and the etiology is labeled idiopathic by default. Uncommonly, an area of demyelination from multiple sclerosis may be the precipitant. Lesions of the entry zone of the trigeminal roots within the pons may cause a similar pain syndrome. Thus, although TN typically is caused by a dysfunction in the peripheral nervous system (the roots or trigeminal nerve itself), a lesion within the central nervous system may rarely cause similar problems. Infrequently, adjacent dental fillings composed of dissimilar metals may trigger attacks. Frequency: Internationally: TN is uncommon, with an estimated prevalence of 155 cases per million persons. Mortality/Morbidity: No mortality is associated with idiopathic TN, although secondary depression is common if a chronic pain syndrome evolves. In rare cases, pain may be so frequent that oral nutrition is impaired. In symptomatic or secondary TN, morbidity or mortality relates to the underlying cause of the pain syndrome. Sex: Male-to-female ratio is 2:3. Age: Development of trigeminal neuralgia in a young person suggests the possibility of multiple sclerosis. Idiopathic TN typically occurs in patients in the sixth decade of life, but it may occur at any age. Symptomatic or secondary TN tends to occur in younger patients. 27 Background: Trigeminal neuralgia (TN), also known as tic douloureux, is a pain syndrome recognizable by patient history alone. The condition is characterized by pain often accompanied by a brief facial spasm or tic. Pain distribution is unilateral and follows the sensory distribution of cranial nerve V, typically radiating to the maxillary (V2) or mandibular (V3) area. At times, both distributions are affected. Physical examination eliminates alternative diagnoses. Signs of cranial nerve dysfunction or other neurologic abnormality exclude the diagnosis of idiopathic TN and suggest that pain may be secondary to a structural lesion. Pathophysiology: The mechanism of pain production remains controversial. One theory suggests that peripheral injury or disease of the trigeminal nerve increases afferent firing in the nerve; failure of central inhibitory mechanisms may be involved as well. Pain is perceived when nociceptive neurons in a trigeminal nucleus involve thalamic relay neurons. Aneurysms, tumors, chronic meningeal inflammation, or other lesions may irritate trigeminal nerve roots along the pons. An abnormal vascular course of the superior cerebellar artery is often cited as the cause. In most cases, no lesion is identified, and the etiology is labeled idiopathic by default. Uncommonly, an area of demyelination from multiple sclerosis may be the precipitant. Lesions of the entry zone of the trigeminal roots within the pons may cause a similar pain syndrome. Thus, although TN typically is caused by a dysfunction in the peripheral nervous system (the roots or trigeminal nerve itself), a lesion within the central nervous system may rarely cause similar problems. Infrequently, adjacent dental fillings composed of dissimilar metals may trigger attacks. http://www.MyTrigeminalNeuralgiaStory.com (Less)
The Litlle Old Lady From Passadena jan & dean Jan and Dean were a rock and roll duo, popular from the late 1950s through the mid 1960s, consisting (More) Jan and Dean were a rock and roll duo, popular from the late 1950s through the mid 1960s, consisting of William Jan Berry (April 3, 1941 March 26, 2004) and Dean Ormsby Torrence (born March 10, 1940). Although Jan & Dean pre-dated The Beach Boys, they became most famously associated with the vocal "surf music" craze inspired by The Beach Boys Jan Berry and Dean Torrence, both born in Los Angeles, California, began singing together as a duo after football practice at University High School. Primitive recording sessions followed soon after, in a makeshift studio in Berry's garage. They first performed onstage as "The Barons" at a high school dance. With the Barons, Jan Berry was experimenting with multi-part vocal arrangements five years before he started working professionally with Brian Wilson.[1] Their first commercial success was "Jennie Lee" (1958), which reached #8, and was an ode to a local, Hollywood burlesque performer that Jan Berry recorded with fellow Baron Arnie Ginsburg. "Jan & Arnie" released three singles in all. After Torrence returned from a stint in the army reserves, Jan Berry and Dean Torrence began to make music as "Jan and Dean". With the help of record producers Herb Alpert and Lou Adler, Jan and Dean scored a #10 hit with "Baby Talk" (1959), and then scored a series of hits over the next couple of years. Playing local venues, they met and performed with the Beach Boys, and discovered the appeal of the latter's "surf sound". By this time, Berry was co-writing, arranging, and producing all of Jan and Dean's original material. Berry signed a series of contracts with Screen Gems to write and produce music for Jan and Dean, as well as other artists such as Judy & Jill (which included Berry's girlfriend Jill Gibson and Dean Torrence's girlfriend Judy Lovejoy), The Matadors, and Pixie (a young female solo singer).[2] During this time, Berry co-wrote and/or arranged and produced songs for artists outside of Jan and Dean, including The Angels ("I Adore Him", Top 30), the Gents, the Matadors (Sinners), Judy & Jill, Pixie (unreleased), Jill Gibson, Shelley Fabares, Deane Hawley, the Rip Chords ("Three Window Coupe", Top 30), and Johnny Crawford, among others. [edit] Part-time musicians Unlike most other rock 'n roll acts of the period, Jan and Dean did not give music their full-time attention. Jan & Dean were college students, maintaining their studies while writing and recording music and making public appearances on the side. Torrence majored in advertising design in the school of architecture at USC. Berry took science and music classes at UCLA, and entered the California College of Medicine (now the UC Irvine School of Medicine) in 1963. By the time of his 1966 auto accident, Berry had completed two years of medical school.[3] [edit] Surf's golden boys: 1963-1964 Jan and Dean reached their commercial peak in 1963 and 1964. The duo scored an impressive sixteen Top 40 hits on the Billboard and Cash Box magazine charts, with a total of twenty-six chart hits over an eight-year period (1958-1966). Jan and Brian Wilson collaborated on roughly a dozen hits and album cuts for Jan and Dean, including the number one national hit "Surf City" in 1963. Subsequent top 10 hits included "Drag City" (#10) (1963), "The Little Old Lady from Pasadena" (#3) (1964), and the eerily portentous "Dead Man's Curve" (#8) (1964). In 1964, at the height of their fame, Jan and Dean hosted and performed at The T.A.M.I. Show, a historic concert film directed by Steve Binder. The film also featured such acts as The Rolling Stones, Chuck Berry, Gerry & The Pacemakers, James Brown, Billy J. Kramer & The Dakotas, Marvin Gaye, The Supremes, Lesley Gore, Smokey Robinson & The Miracles, and the Beach Boys (whose sequence was later cut from the film, due to contract violation issues). Also in 1964, the duo performed the title track for the Columbia Pictures film Ride the Wild Surf, starring Fabian, Tab Hunter, Peter Brown, Shelley Fabares, and Barbara Eden. The song, penned by Jan Berry, Brian Wilson, and Roger Christian, was a Top 20 national hit. Jan and Dean also filmed two unreleased television pilots: Surf Scene in 1963 and On the Run in 1966. Their feature film Easy Come, Easy Go was canceled when Berry, as well as the film's director and other crew members, were seriously injured in a railroad accident while shooting the movie in August 1965. [edit] Changing times: 1965-1966 After the surf craze, Jan and Dean scored two Top-30 hits in 1965: "You Really Know How to Hurt a Guy" and "I Found a Girl" the latter from the album Folk 'n Roll. During this period, they also began to experiment with cutting-edge comedy concepts such as the original (unreleased) Filet of Soul and Jan & Dean Meet Batman. The former's album cover shows Berry with his leg in a cast as a result of the accident while filming "Easy Come, Easy Go". [edit] Berry's car wreck and its aftermath: 1966-1968 On April 12, 1966, Berry received severe head injuries in a motor vehicle accident just a short distance from Dead Man's Curve in Los Angeles, California, two years after the song had become a hit. Berry was on his way to a business meeting when he crashed his Corvette into a parked truck on Whittier Drive in Beverly Hills. Berry had also separated from his girlfriend of seven years, singer-artist Jill Gibson, later a member for a short time of The Mamas and the Papas, who had also co-written several songs with Berry. Berry traveled a long and difficult road toward recovery from brain damage and partial paralysis. He had minimal use of his right arm, and had to learn to write with his left hand. Doctors said he would never walk again, but he refused to give up, and ultimately succeeded. Torrence stood by his partner, maintaining their presence in the music industry, and keeping open the possibility that they would perform together again.[4] In Berry's absence, Torrence released several singles on the J&D Record Co. label and recorded Save for a Rainy Day in 1966, a concept album featuring all rain-themed songs. Torrence posed with Berry's brother Ken for the album cover photos. Columbia Records released one single from the project ("Yellow Balloon") as did the song's writer, Gary Zekley, with The Yellow Balloon, but with legal wrangles scuttling Torrence's Columbia deal and Berry's disapproval of the project, Save for a Rainy Day remained a self-released album on the J&D Record Co. label.[5] Besides his studio work, Torrence became a graphic artist while Berry recovered, starting his own company, Kittyhawk Graphics, and designing and creating album covers and logos for other musicians and recording artists, including Harry Nilsson, Steve Martin, the Nitty Gritty Dirt Band, Dennis Wilson, Bruce Johnston, The Beach Boys, Diana Ross and The Supremes, Linda Ronstadt, Papa Doo Run Run, Canned Heat, The Ventures and many others. Torrence (with Gene Brownell) won a Grammy Award for Album Cover of the Year, for the group Pollution in 1973. Berry returned to the studio in April 1967, one year to the month after his accident. Working with collaborators, he began writing and producing music again. In December 1967, Jan and Dean signed an agreement with Warner Bros. Records. Warner issued two singles under the name Jan and Dean, but a 1968 Berry-produced album for Warner Bros., the psychedelic Carnival of Sound, remains unreleased.[6] [edit] Further progress: 1969-1978 Berry began to sing again in the early 1970s, and he arranged and produced a number of singles (both solo and as Jan & Dean) between 1972 and 1978 on the Ode and A&M labels, facilitated by friend and former manager Lou Adler.[7]Berry also toured with his Aloha band, while Dean began performing with a band called Papa Doo Run Run. In 1973, Jan & Dean made an appearance at the Hollywood Palladium, as part of Jim Pewter's "Surfer's Stomp" reunion. But the duo's first performance after Berry's accident backed with live musicians occurred at the Palomino Nightclub in North Hollywood, June 5, 1976 (ten years after the accident). Backing the duo was Dean's band, Papa Doo Run Run. The day after that performance there was a very positive review in "Variety" and the phones started ringing. Promoters everywhere wanted to hire Jan & Dean. So in the fall of 1976 a mini-tour of the Pacific Northwest was planned with Jan & Dean backed by Papa Doo Run Run. The tour was a rousing success. J & D then hired a manager and agency representation, and between 1977 and 1980, there were 4 nationwide J & D tours with Papa Doo as their opening act and backup band - sell out crowds from coast to coast. Even though a dozen years had passed, Jan was still suffering the effects of his horrific accident, with partial paralysis and aphasia. But Jan's limp and useless right arm, plus his difficulty in speaking were totally overlooked by his adoring fans. Jan Berry was back! [8] [9] In 1974, attorney Paul Morantz published a landmark article about Jan Berry's recovery in Rolling Stone magazine.[10] [edit] Back on the road: 1978-2004 On February 3, 1978, CBS aired a made-for-TV movie about the duo titled Deadman's Curve. The biopic starred Richard Hatch as Jan Berry and Bruce Davison as Dean Torrence, with cameo appearances by Dick Clark, Wolfman Jack, Mike Love of the Beach Boys, and Bruce Johnston (who at that time was temporarily out of the Beach Boys), as well as Berry himself (near the end of the movie, he can be seen sitting in the audience, watching "himself" (Richard Hatch) perform onstage). The part of Jan & Dean's band, Papa Doo Run Run, was played by themselves. Johnston and Berry had known each other since high school, and had played music together in Berry's garage in Bel Air long before Jan & Dean or the Beach Boys were formed. Following the release of the film, the duo made steps toward an official comeback that year, including touring with the Beach Boys. In the early 1980s, Papa Doo Run Run left the duo to explore other performance and recording ventures. Berry struggled to overcome drug addiction, so Torrence toured briefly as "Mike & Dean", with Mike Love of the Beach Boys. Once Berry got sober, beating the odds once again, the duo reunited for good. In "Phase II" of their career, Dean Torrence led the touring operation. In 1986, Berry helped establish the Jan Berry Center for the Brain Injured in Downey, California. Dean Torrence participated in the promotional campaign for this endeavor. Though Berry only made a partial recovery, he persevered and remained a high-profile example for patients with traumatic brain injury.[11] Jan and Dean continued to tour on their own throughout the 1980s, 1990s, and into the new millennium with 1960s nostalgia providing them with a ready audience. Sundazed Records reissued Save for a Rainy Day in 1996, and the album drew critical praise.[citation needed] Between the 1970s and 1990s, Torrence issued a number of re-recordings of classic Jan and Dean hits. An album titled One Summer Night / Live was issued by Rhino Records in 1982, and Dean collaborated with Berry on Port to Paradise, released on J&D Records in 1986. In 1997, after many years of hard work, Berry released a solo album called Second Wave on One Way Records. On August 31, 1991, Berry married Gertie Filip at The Stardust Convention Centre in Las Vegas, Nevada. Torrence was Berry's best man at the wedding. Jan and Dean ended with Jan Berry's death on March 26, 2004, at the age of 62. Berry was an organ donor, and his body was cremated. On April 18, 2004, a "Celebration of Life" was held in Berry's memory at The Roxy Theatre on the Sunset Strip in West Hollywood, California. Celebrities attending the event included Dean Torrence, Lou Adler, Jill Gibson, and Nancy Sinatra. Also present were many family members, friends, and musicians associated with Jan and Dean and the Beach Boys including the original 1970's version of Papa Doo Run Run. Torrence now tours occasionally with The Surf City Allstars. He serves as a spokesman for the City of Huntington Beach California, which, thanks in part to his efforts, is nationally recognized as "Surf City USA". He officially endorses the Official Jan & Dean Fan Site. Dean Torrence lives in Huntington Beach, California with his wife and two daughters. Katie Torrence, his oldest daughter, is reportedly recording an album.[citation needed] [edit] Jan and Dean's place in rock history According to rock critic Dave Marsh, the attitude and public persona of punk rock can be traced to Jan and Dean.[12] Moreover, both Jan Berry and Dean Torrence's anti-establishment attitude toward the music industry is well documented.[citation needed] Their music has been covered by numerous Punk and alternative bands since the 1970s. Along with Phil Spector, Brian Wilson, and Lee Hazlewood, Berry enjoyed a reputation as one of the best record producers on the West Coast.[13] Brian Wilson has cited Berry as having a direct impact on his own growth as a record producer.[14] Dean Torrence believes the group should be in the Rock & Roll Hall of Fame: "We have the scoreboard if you just want to compare number of hits and musical projects done. We beat 75 percent of the people in there. So what else is it? I've got to think that we were pretty irreverent when it came to the music industry. They kind of always held that against us. That's OK with me."[ (Less)
3-6 Microvascular Decompression MVD Dr. Parrish Neurosurgeon Click More http://www.MyTrigeminalNeuralgiaStory.com AWC 4398 3-6 Microvascular Decompression MVD (More) Click More http://www.MyTrigeminalNeuralgiaStory.com AWC 4398 3-6 Microvascular Decompression MVD Click Dr.Parrish Neurosurgeon TN Tic douloureux Facial Pain Electric Shocks. TNA BrianNelson123 Suicide Painful Jannetta Association Teflon Nerve THIS WEBSITE IS DESIGNED TO HAVE EACH TRIGEMINAL NEURALGIA patient tell there story from the beginning of the problem to the current status which is understandably changing daily as the body processes more of the pain. My personal story is very long and and be seen at w htttp[://www.IamFightingCancer.com Important words found on this site. Trigeminal Neuralgia Minneapolis TN Pain Personal Story, Balloon Compression Mentor, dysesthesia, bad feeling constant spasm. excruciating pains, Henry, Pneumonia Electrical Shocks, Shirley, Shelly Wilson, Support Group, Education, Association, Stabbing, Jolts, Suicide Disease, Neuropathic, rare Disorder, Treatment, destructive surgery, Procedure, Microvascular Decompression, tic douloureux Marge Prietz Trigeminal Neuralgia Extreme Facial Pain TN Websites insert. YouTube. From NelsonIdeas.com Trigeminal Neuralgia Extreme Facial Pain TN Websites insert. Websites insert. My Trigeminal Neuralgia Extreme Facial Pain TN Websites http:/./www.NelsonIdeas.com Click Dental Education Trigeminal Neuralgia Extreme Facial Pain http://www.NewMedicalDirectories.com/Trigeminal-Neuralgia-Dental/Dentist-Dentists.html Click Trigeminal Neuralgia Patient Painful-Stories http://www.BrianNelsonConsulting.com/trigeminal-neuralgia-tn/patient-painful-stories.html Click My Trigeminal Neuralgia (TN) Story only http://www.PartyTentCity.com/mytnstory.html Click My Story on TN Brian N http://www.PartyTentCity.com/trigeminal-neuralgia-tn-tmj-my-story/directory.html Click Trigeminal Neuralgia Slide Show Story of Pain http://www.NewMedicalDirectories.com/Trigeminal-Neuralgia-Slide-Show/Draft.html Click Medical Data Base Medical Costs More Expensive Due to Non Use of Technology http://www.briannelsonconsulting.com/medical-data-base/faq-info.html Click MyTrigeminal Neuralgia Story Directory http://www.MyTrigeminalNeuralgiaStory.com Click Slide Show Draft for New TN Patients. http://www.newmedicaldirectories.com/Trigeminal-Neuralgia-Slide-Show/Draft.html Click-Trigeminal Neuralgia Assn Page 1 http://newmedicaldirectories.com/Trigeminal-Neuralgia-Association/TN-Facial-Pain.html Click-Trigeminal Neuralgia Assn Page 2 http://newmedicaldirectories.com/Trigeminal-Neuralgia-Association/TN-Facial-Pain-2.html Click What is Trigeminal Neuragia? Portland,OR Slide Show http://www.NewMedicalDirectories.com/Trigeminal-Neuralgia-Slide-Show/Draft.html Click Trigeminal Neuralgia National Conference http://www.NewMedicalDirectories.com/Trigeminal-Neuralgia-Slide-Show/Draft.html Click Trigeminal Neuralgia Brian's Journal Tic Douloureux (TN) FacialPain-Cancer http://www.BrianNelsonConsulting.com/trigeminal-neuralgia-tn/faq-info.html http://www.BrianNelsonConsulting.com/trigeminal-neuralgia-tn/faq-info.html Click Page 1. Trigeminal Neuralgia http://www.BrianNelsonConsulting.com/trigeminal-neuralgia-tn/faq-info.html Click Page 2 Trigeminal Neuralgia http://www.briannelsonconsulting.com/trigeminal-neuralgia-tn/faq-info2.html Click Page 3 Trigeminal Neuralgia http://www.briannelsonconsulting.com/trigeminal-neuralgia-tn/faq-info3.htm Click Page 4 Trigeminal Neuralgia http://www.BrianNelsonConsulting.com/trigeminal-neuralgia-tn/faq-info4.html Click MyTrigeminal Neuralgia Stories Directory http://www.MyTrigeminalNeuralgiaStory.com/Index.html Click Brian's TN Story Quck Version http://www.MyTrigeminalNeuralgiaStory.com/BrianNelson/TN1.html Click Shirley's Story Trigeminal Neuralgia http://www.MyTrigeminalNeuralgiaStory.com/ShirleyH/TN3.html Click Sand's Story TN WHAT IS TRIGEMINAL NEURALGIA? TN (Trigeminal Neuralgia) is a pain that is described as among the most acute known to mankind. TN produces excruciating, lightning strikes of facial pain, typically near the nose, lips, eyes or ears. It is a disorder of the trigeminal nerve, which is the fifth and largest cranial nerve. TN (Trigeminal Neuralgia / tic douloureux) is a disorder of the fifth cranial (trigeminal) nerve that causes episodes of intense, stabbing, electric shock-like pain in the areas of the face where the branches of the nerve are distributed - lips, eyes, nose, scalp, forehead, upper jaw, and lower jaw. By many, it's called the "suicide disease". A less common form of the disorder called "Atypical Trigeminal Neuralgia" may cause less intense, constant, dull burning or aching pain, sometimes with occasional electric shock-like stabs. Both forms of the disorder most often affect one side of the face, but some patients experience pain at different times on both sides. Onset of symptoms occurs most often after age 50, but cases are known in children and even infants. Something as simple and routine as brushing the teeth, putting on makeup or even a slight breeze can trigger an attack, resulting in sheer agony for the individual. Trigeminal neuralgia (TN) is not fatal, but it is universally considered to be the most painful affliction known to medical practice. Initial treatment of TN is usually by means of anti-convulsant drugs, such as Tegretol or Neurontin. Some anti-depressant drugs also have significant pain relieving effects. Should medication be ineffective or if it produces undesirable side effects, neurosurgical procedures are available to relieve pressure on the nerve or to reduce nerve sensitivity. Some patients report having reduced or relieved pain by means of alternative medical therapies such as acupuncture, chiropractic adjustment, self-hypnosis or meditation. http://www.MyTrigeminalNeuralgiaStory.com/SandiW/TN4.html What is Trigeminal Neuralgia? Trigeminal neuralgia (TN), also called tic douloureux, is a chronic pain condition that causes extreme, sporadic, sudden burning or shock-like face pain that lasts anywhere from a few seconds to as long as 2 minutes per episode. The intensity of pain can be physically and mentally incapacitating. TN pain is typically felt on one side of the jaw or cheek. Episodes can last for days, weeks, or months at a time and then disappear for months or years. In the days before an episode begins, some patients may experience a tingling or numbing sensation or a somewhat constant and aching pain. The attacks often worsen over time, with fewer and shorter pain-free periods before they recur. The intense flashes of pain can be triggered by vibration or contact with the cheek (such as when shaving, washing the face, or applying makeup), brushing teeth, eating, drinking, talking, or being exposed to the wind. TN occurs most often in people over age 50, but it can occur at any age, and is more common in women than in men. There is some evidence that the disorder runs in families, perhaps because of an inherited pattern of blood vessel formation. Although sometimes debilitating, the disorder is not life-threatening. The presumed cause of TN is a blood vessel pressing on the trigeminal nerve in the head as it exits the brainstem. TN may be part of the normal aging process but in some cases it is the associated with another disorder, such as multiple sclerosis or other disorders characterized by damage to the myelin sheath that covers certain nerves. Is there any treatment? Because there are a large number of conditions that can cause facial pain, TN can be difficult to diagnose. But finding the cause of the pain is important as the treatments for different types of pain may differ. Treatment options include medicines such as anticonvulsants and tricyclic antidepressants, surgery, and complementary approaches. Typical analgesics and opioids are not usually helpful in treating the sharp, recurring pain caused by TN. If medication fails to relieve pain or produces intolerable side effects such as excess fatigue, surgical treatment may be recommended. Several neurosurgical procedures are available. Some are done on an outpatient basis, while others are more complex and require hospitalization. Some patients choose to manage TN using complementary techniques, usually in combination with drug treatment. These techniques include acupuncture, biofeedback, vitamin therapy, nutritional therapy, and electrical stimulation of the nerves. What is the prognosis? The disorder is characterized by recurrences and remissions, and successive recurrences may incapacitate the patient. Due to the intensity of the pain, even the fear of an impending attack may prevent activity. Trigeminal neuralgia is not fatal. What research is being done? Within the NINDS research programs, trigeminal neuralgia is addressed primarily through studies associated with pain research. NINDS vigorously pursues a research program seeking new treatments for pain and nerve damage with the ultimate goal of reversing debilitating conditions such as trigeminal neuralgia. NINDS has notified research investigators that it is seeking grant applications both in basic and clinical pain research. An Alternate Strategy Instead of waiting for the pain to become intractable or the medications toxic, an individual with trigeminal neuralgia has the option to request early surgery. This has a number of potential advantages: • Avoid years of medication and intermittent pain • Avoid facing surgery when old or infirm • If the person has a vascular loop, early microvascular decompression will increase the possibility of a successful operation with decreased risk of recurrence (evidence suggests better outcomes and lower recurrence rate the shorter the interval between onset of symptoms and nerve decompression) How To Find Out If You Have a Vascular Loop The conventional MRI scans used to rule out the presence of a brain tumor or multiple sclerosis as a cause of a patients face pain are not adequate to visualize the trigeminal nerve or an associated blood vessel. Fortunately, the continued improvement in MRI neuro-imaging now makes it possible to visualize both. The technique, which is called 3-D volume acquisition, is performed with contrast injection and utilizes thin cuts (0.8mm), without gaps similar to what was developed for MRI angiography and venography. The trigeminal nerve is easily visualized in the axial plane when the MRI series is centered at the midpoint of the fourth ventricle. To ensure an adequate evaluation, the nerve should be seen on three adjacent cuts. Early studies indicate that when an offending vessel is present it will be detected 80% of the of the time. With continued imaging improvements this percentage will definitely increase. Click here for UCSD Trigeminal Neuralgia Sequence Parameters for Seimens and GE MR Scanners. Surgical Options: Non-Destructive Procedures The only non-destructive procedure which reliably relieves the symptoms of Trigeminal Neuralgia is Microvascular Decompression (MVD). This involves surgical exploration with the operating microscope and visualization of the junction where the Trigeminal nerve enters the base of the brain, followed by coagulation or moving and padding away any compressing blood vessels. The advantage is pain relief without numbness in the majority of patients, which usually lasts indefinitely. If the pain recurs after a MVD, which it does in 10-15% of patients, it can usually be controlled with low dose Tegretol® or Neurontin®. If the pain continues, it will require a repeat MVD or one of the destructive procedures. Surgical Options: Destructive Procedures There are multiple destructive procedures which are beneficial in the treatment of Trigeminal Neuralgia. The most common of which are glycerol injections, gamma knife radiation, electrocoagulation, and balloon compression. These procedures are all based on interrupting the pain by partial damage to Trigeminal nerve fibers. Generally the more numbness they produce, the longer they last. The specific advantages and disadvantages need to be discussed with the surgeon performing the procedure. These procedures are recommended for patients who have failed MVD or are not candidates for major surgery. Comments Treatment is always individualized. All of the options above should be considered in consultation with a neurosurgeon familiar in their use. Recommendations Based on the data currently available, and in an effort to maximize quality of life, we recommend the following: Patients with less than 10 year life expectancy Refer for destructive procedure if pain not controlled medically without significant side effects Patients with more than 10 but less than 20 year life expectancy Consider destructive procedure May abolish need for continued increasing medications Will make medical therapy easier even if fails Patients with more than 20 year life expectancy Perform thin cut MRI with 3-D Volume Acquisition If vessel present recommend MVD 25 ARTICLE SECTIONS From the Mayo Clinic. Trigeminal neuralgia http://www.mayoclinic.com/health/trigeminal-neuralgia/DS00446 Introduction Signs and symptoms Causes When to seek medical advice Screening and diagnosis Treatment Coping skills Introduction Imagine having a jab of lightning-like pain shoot through your face when you brush your teeth or put on makeup. Sound excruciating? If you have trigeminal neuralgia, attacks of such pain are frequent and can often seem unbearable. You may initially experience short, mild attacks, but trigeminal neuralgia can progress, causing longer, more frequent bouts of searing pain. These painful attacks can be spontaneous, but they may also be provoked by even mild stimulation of your face, including brushing your teeth, shaving or putting on makeup. The pain of trigeminal neuralgia may occur in a fairly small area of your face, or it may spread rapidly over a wider area. Because of the variety of treatment options available, having trigeminal neuralgia doesn't necessarily mean you're doomed to a life of pain. Doctors usually can effectively manage trigeminal neuralgia, either with medications or surgery. Signs and symptoms An attack of trigeminal neuralgia can last from a few seconds to about a minute. Some people have mild, occasional twinges of pain, while other people have frequent, severe, electric-shock-like pain. The condition tends to come and go. You may experience attacks of pain off and on all day, or even for days or weeks at a time. Then, you may experience no pain for a prolonged period of time. Remission is less common the longer you have trigeminal neuralgia. People who have experienced severe trigeminal neuralgia have described the pain as: Lightning-like or electric-shock-like Shooting Jabbing Like having live wires in your face Trigeminal neuralgia usually affects just one side of your face. The pain may affect just a portion of one side of your face or spread in a wider pattern. Rarely, trigeminal neuralgia can affect both sides of your face, but not at the same time. Causes Branches of the trigeminal nerve CLICK TO ENLARGE The condition is called trigeminal neuralgia because the painful facial areas are those served by one or more of the three branches of your trigeminal nerve. This large nerve originates deep inside your brain and carries sensation from your face to your brain. The pain of trigeminal neuralgia is due to a disturbance in the function of the trigeminal nerve. Trigeminal neuralgia is also known as tic douloureux. The cause of the pain usually is due to contact between a normal artery or vein and the trigeminal nerve at the base of your brain. This places pressure on the nerve as it enters your brain and causes the nerve to misfire. Physical nerve damage or stress may be the initial trigger for trigeminal neuralgia. After the trigeminal nerve leaves your brain and travels through your skull, it divides into three smaller branches, controlling sensation throughout your face: The first branch controls sensation in your eye, upper eyelid and forehead. The second branch controls sensation in your lower eyelid, cheek, nostril, upper lip and upper gum. The third branch controls sensations in your jaw, lower lip, lower gum and some of the muscles you use for chewing. You may feel pain in the area served by just one branch of the trigeminal nerve, or the pain may affect all branches on one side of your face. Besides compression from blood vessel contact, other less frequent sources of pain to the trigeminal nerve may include: Compression by a tumor Multiple sclerosis A stroke affecting the lower part of your brain, where the trigeminal nerve enters your central nervous system A variety of triggers, many subtle, may set off the pain. These triggers may include: Shaving Stroking your face Eating Drinking Brushing your teeth Talking Putting on makeup Encountering a breeze Smiling Trigeminal neuralgia affects women more often than men. The disorder is more likely to occur in people who are older than 50. About 5 percent of people with trigeminal neuralgia have other family members with the disorder, which suggests a possible genetic cause in some cases. When to seek medical advice Some people mistake the pain of trigeminal neuralgia for a toothache or a headache. It's not uncommon for people to believe that their facial pain is dental-related, particularly when the pain seems to stem from the gumline or is located near a tooth. If you experience facial pain, particularly prolonged pain or pain that hasn't gone away with use of over-the-counter pain relievers, see your dentist or doctor. Screening and diagnosis If you go to your dentist, an examination of your mouth can reveal whether a problem with your teeth or gums is causing your pain. If you go to your doctor, he or she will want to ask about your medical history and have you describe your pain — how severe it is, what part of your face it affects, how long pain lasts and what seems to trigger episodes of pain. You'll also undergo a neurologic examination. During this examination, your doctor examines and touches parts of your face to try to determine exactly where the pain is occurring and — if it appears that you have trigeminal neuralgia — which branches of the trigeminal nerve may be affected. Your doctor may exclude other possible conditions based on your medical history, the examination, and a magnetic resonance imaging (MRI) scan of your head. Treatment Medications are the usual initial treatment for trigeminal neuralgia. Medications are often effective in lessening or blocking the pain signals sent to your brain. A number of drugs are available. If you stop responding to a particular medication or experience too many side effects, switching to another medication may work for you. Medications Carbamazepine (Tegretol, Carbatrol). Carbamazepine, an anticonvulsant drug, is the most common medication that doctors use to treat trigeminal neuralgia. In the early stages of the disease, carbamazepine controls pain for most people. However, the effectiveness of carbamazepine decreases over time. Side effects include dizziness, confusion, sleepiness and nausea. Baclofen. Baclofen is a muscle relaxant. Its effectiveness may increase when it's used in combination with carbamazepine or phenytoin. Side effects include confusion, nausea and drowsiness. Phenytoin (Dilantin, Phenytek). Phenytoin, another anticonvulsant medication, was the first medication used to treat trigeminal neuralgia. Side effects include gum enlargement, dizziness and drowsiness. Oxcarbazepine (Trileptal). Oxcarbazepine is another anticonvulsant medication and is similar to carbamazepine. Side effects include dizziness and double vision. Doctors may sometimes prescribe other medications, such as lamotrignine (Lamictal) or gabapentin (Neurontin). Some people with trigeminal neuralgia eventually stop responding to medications, or they experience unpleasant side effects. For those people, surgery, or a combination of surgery and medications, may be an option. Surgery The goal of a number of surgical procedures is to either damage or destroy the part of the trigeminal nerve that's the source of your pain. Because the success of these procedures depends on damaging the nerve, facial numbness of varying degree is a common side effect. These procedures involve: Alcohol injection. Alcohol injections under the skin of your face, where the branches of the trigeminal nerve leave the bones of your face, may offer temporary pain relief by numbing the areas for weeks or months. Because the pain relief isn't permanent, you may need repeated injections or a different procedure. Glycerol injection. This procedure is called percutaneous glycerol rhizotomy (PGR). "Percutaneous" means through the skin. Your doctor inserts a needle through your face and into an opening in the base of your skull. The needle is guided into the trigeminal cistern, a small sac of spinal fluid that surrounds the trigeminal nerve ganglion (the area where the trigeminal nerve divides into three branches) and part of its root. Images are made to confirm that the needle is in the proper location. After confirming the location, your doctor injects a small amount of sterile glycerol. After three or four hours, the glycerol damages the trigeminal nerve and blocks pain signals. Initially, PGR relieves pain in most people. However, some people have a recurrence of pain, and many experience facial numbness or tingling. http://www.MyTrigeminalNeuralgiaStory.com Balloon compression. In a procedure called percutaneous balloon compression of the trigeminal nerve (PBCTN), your doctor inserts a hollow needle through your face and into an opening in the base of your skull. Then, a thin, flexible tube (catheter) with a balloon on the end is threaded through the needle. The balloon is inflated with enough pressure to damage the nerve and block pain signals. PBCTN successfully controls pain in most people, at least for a while. Most people undergoing PBCTN experience facial numbness of varying degrees, and more than half experience nerve damage resulting in a temporary or permanent weakness of the muscles used to chew. http://www.MyTrigeminalNeuralgiaStory.com Electric current. A procedure called percutaneous stereotactic radiofrequency thermal rhizotomy (PSRTR) selectively destroys nerve fibers associated with pain. Your doctor threads a needle through your face and into an opening in your skull. Once in place, an electrode is threaded through the needle until it rests against the nerve root. An electric current is passed through the tip of the electrode until it's heated to the desired temperature. The heated tip damages the nerve fibers and creates an area of injury (lesion). If your pain isn't eliminated, your doctor may create additional lesions. PSRTR successfully controls pain in most people. Facial numbness is a common side effect of this type of treatment. The pain may return after a few years. Microvascular decompression (MVD). A procedure called microvascular decompression (MVD) doesn't damage or destroy part of the trigeminal nerve. Instead, MVD involves relocating or removing blood vessels that are in contact with the trigeminal root and separating the nerve root and blood vessels with a small pad. During MVD, your doctor makes an incision behind one ear. Then, through a small hole in your skull, part of your brain is lifted to expose the trigeminal nerve. If your doctor finds an artery in contact with the nerve root, he or she directs it away from the nerve and places a pad between the nerve and the artery. Doctors usually remove a vein that is found to be compressing the trigeminal nerve. MVD can successfully eliminate or reduce pain most of the time, but as with all other surgical procedures for trigeminal neuralgia, pain can recur in some people. http://www.MyTrigeminalNeuralgiaStory.com While MVD has a high success rate, it also carries risks. There are small chances of decreased hearing, facial weakness, facial numbness, double vision, and even a stroke or death. The risk of facial numbness is less with MVD than with procedures that involve damaging the trigeminal nerve. Severing the nerve. A procedure called partial sensory rhizotomy (PSR) involves cutting part of the trigeminal nerve at the base of your brain. Through an incision behind your ear, your doctor makes a quarter-sized hole in your skull to access the nerve. This procedure usually is helpful, but almost always causes facial numbness. And it's possible for pain to recur. If your doctor doesn't find an artery or vein in contact with the trigeminal nerve, he or she won't be able to perform an MVD, and a PSR may be done instead. Radiation. Gamma-knife radiosurgery (GKR) involves delivering a focused, high dose of radiation to the root of the trigeminal nerve. The radiation damages the trigeminal nerve and reduces or eliminates the pain. Relief isn't immediate and can take several weeks to begin. GKR is successful in eliminating pain more than half of the time. Sometimes the pain may recur. The procedure is painless and typically is done without anesthesia. Because this procedure is relatively new, the long-term risks of this type of radiation are not yet known. • Coping skills Living with trigeminal neuralgia can be difficult. The disorder may affect your interaction with friends and family, your productivity at work, and the overall quality of your life. You may find that talking to a counselor or therapist can help you cope with the effects of trigeminal neuralgia, or you may find encouragement and understanding in a support group. Although support groups aren't for everyone, they can be good sources of information. Group members often know about the latest treatments and tend to share their own experiences. If you're interested, your doctor may be able to recommend a group in your area. 27 Background: Trigeminal neuralgia (TN), also known as tic douloureux, is a pain syndrome recognizable by patient history alone. The condition is characterized by pain often accompanied by a brief facial spasm or tic. Pain distribution is unilateral and follows the sensory distribution of cranial nerve V, typically radiating to the maxillary (V2) or mandibular (V3) area. At times, both distributions are affected. Physical examination eliminates alternative diagnoses. Signs of cranial nerve dysfunction or other neurologic abnormality exclude the diagnosis of idiopathic TN and suggest that pain may be secondary to a structural lesion. Pathophysiology: The mechanism of pain production remains controversial. One theory suggests that peripheral injury or disease of the trigeminal nerve increases afferent firing in the nerve; failure of central inhibitory mechanisms may be involved as well. Pain is perceived when nociceptive neurons in a trigeminal nucleus involve thalamic relay neurons. Aneurysms, tumors, chronic meningeal inflammation, or other lesions may irritate trigeminal nerve roots along the pons. An abnormal vascular course of the superior cerebellar artery is often cited as the cause. In most cases, no lesion is identified, and the etiology is labeled idiopathic by default. Uncommonly, an area of demyelination from multiple sclerosis may be the precipitant. Lesions of the entry zone of the trigeminal roots within the pons may cause a similar pain syndrome. Thus, although TN typically is caused by a dysfunction in the peripheral nervous system (the roots or trigeminal nerve itself), a lesion within the central nervous system may rarely cause similar problems. Infrequently, adjacent dental fillings composed of dissimilar metals may trigger attacks. Frequency: Internationally: TN is uncommon, with an estimated prevalence of 155 cases per million persons. Mortality/Morbidity: No mortality is associated with idiopathic TN, although secondary depression is common if a chronic pain syndrome evolves. In rare cases, pain may be so frequent that oral nutrition is impaired. In symptomatic or secondary TN, morbidity or mortality relates to the underlying cause of the pain syndrome. Sex: Male-to-female ratio is 2:3. Age: Development of trigeminal neuralgia in a young person suggests the possibility of multiple sclerosis. Idiopathic TN typically occurs in patients in the sixth decade of life, but it may occur at any age. Symptomatic or secondary TN tends to occur in younger patients. 27 Background: Trigeminal neuralgia (TN), also known as tic douloureux, is a pain syndrome recognizable by patient history alone. The condition is characterized by pain often accompanied by a brief facial spasm or tic. Pain distribution is unilateral and follows the sensory distribution of cranial nerve V, typically radiating to the maxillary (V2) or mandibular (V3) area. At times, both distributions are affected. Physical examination eliminates alternative diagnoses. Signs of cranial nerve dysfunction or other neurologic abnormality exclude the diagnosis of idiopathic TN and suggest that pain may be secondary to a structural lesion. Pathophysiology: The mechanism of pain production remains controversial. One theory suggests that peripheral injury or disease of the trigeminal nerve increases afferent firing in the nerve; failure of central inhibitory mechanisms may be involved as well. Pain is perceived when nociceptive neurons in a trigeminal nucleus involve thalamic relay neurons. Aneurysms, tumors, chronic meningeal inflammation, or other lesions may irritate trigeminal nerve roots along the pons. An abnormal vascular course of the superior cerebellar artery is often cited as the cause. In most cases, no lesion is identified, and the etiology is labeled idiopathic by default. Uncommonly, an area of demyelination from multiple sclerosis may be the precipitant. Lesions of the entry zone of the trigeminal roots within the pons may cause a similar pain syndrome. Thus, although TN typically is caused by a dysfunction in the peripheral nervous system (the roots or trigeminal nerve itself), a lesion within the central nervous system may rarely cause similar problems. Infrequently, adjacent dental fillings composed of dissimilar metals may trigger attacks. http://www.MyTrigeminalNeuralgiaStory.com (Less)
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